pre-cteappv Oops 090713

This is a pre-cteappv Oops and I start wondering how Fleury manage to find journals with so poor review process as to let go to press such mistakes.

This one is from An Elasto-Plastic Model of Avian Gastrulation, Vincent Fleury, Organogenesis 2:1, 6-16, 2005.

[p13, col1, §4]

The extraordinary fact is that this equation is exactly the equation for two magnetic dipoles located at +a and -a near the caudal pole.8

Where 8 is given as: Bershadsky AD, Balaban NQ, Geiger B. Adhesion-dependent cell mechanosensitivity, Annual rev. oc Cell and Dev Biol 2003; 19:677-95.

OK, there is a small typo. But that’s not the problem.

Bershadsky et al. paper have just nothing to do with “two magnetic dipoles”! or any “caudal pole”. This is cell biology they are discussing there (abstract and full reference below).

Fast check:

  • equation, absent,
  • magnetic, absent,
  • dipole, absent,
  • caudal, absent,
  • pole, absent

Slow check, I read the paper, nothing that could be confused with Fleury’s statement.

There are just 12 references in that paper, the first one I’m checking is wrong.

So,

The extraordinary fact is that this equation is exactly the equation for two magnetic dipoles located at +a and -a near the caudal pole.8

Either the reference is wrong or Fleury’s assertion. Or both.


Adhesion-Dependent Cell Mechanosensitivity

Alexander D. Bershadsky, Nathalie Q. Balaban, and Benjamin Geiger

Annual Review of Cell and Developmental Biology Vol. 19: 677-695 2003, doi:10.1146/annurev.cellbio.19.111301.153011

The conversion of physical signals, such as contractile forces or external mechanical perturbations, into chemical signaling events is a fundamental cellular process that occurs at cell–extracellular matrix contacts, known as focal adhesions. At these sites, transmembrane integrin receptors are associated via their cytoplasmic domains with the actin cytoskeleton. This interaction with actin is mediated by a submembrane plaque, consisting of numerous cytoskeletal and signaling molecules. Application of intrinsic or external forces to these structures dramatically affects their assembly and triggers adhesion-mediated signaling. In this review, we discuss the structure-function relationships of focal adhesions and the possible mode of action of the putative mechanosensor associated with them. We also discuss the general phenomenon of mechanosensitivity, and the approaches used to measure local forces at adhesion sites, the cytoskeleton-mediated regulation of local contractility, and the nature of the signaling networks that both affect contractility and are affected by it.

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